Circulation Research -- Table of Contents (82 [11])

Volume 82, Issue 11; June 15, 1998

Editorials
Reviews
Original Contributions
Rapid Communications

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	Editorials
	Myocyte Cell Death in the Diseased Heart Piero Anversa and Jan Kajstura Circ Res. 1998;82:1231-1233 Extract \| Full Text \| PDF
	Reviews
	Apoptosis : Basic Mechanisms and Implications for Cardiovascular Disease Armin Haunstetter and Seigo Izumo Circ Res. 1998;82:1111-1129 Extract \| Full Text \| PDF
	Original Contributions
	Role of Apoptosis in the Disappearance of Infiltrated and Proliferated Interstitial Cells After Myocardial Infarction Genzou Takemura, Michiya Ohno, Yukihiro Hayakawa, Jun Misao, Motoo Kanoh, Atsuko Ohno, Yoshihiro Uno, Shinya Minatoguchi, Takako Fujiwara, and Hisayoshi Fujiwara Circ Res. 1998;82:1130-1138 Abstract \| Full Text \| PDF
	Effect of Vacuolar Proton ATPase on pH_i, Ca²⁺, and Apoptosis in Neonatal Cardiomyocytes During Metabolic Inhibition/Recovery Ewa Karwatowska-Prokopczuk, Judith A. Nordberg, Hai Ling Li, Robert L. Engler, and Roberta A. Gottlieb Circ Res. 1998;82:1139-1144 Abstract \| Full Text \| PDF
	Angiotensin II Stimulation In Vitro Induces Hypertrophy of Normal and Postinfarcted Ventricular Myocytes Yu Liu, Annarosa Leri, Baosheng Li, Xiaowei Wang, Wei Cheng, Jan Kajstura, and Piero Anversa Circ Res. 1998;82:1145-1159 Abstract \| Full Text \| PDF
	ß₁ Integrins Participate in the Hypertrophic Response of Rat Ventricular Myocytes Robert S. Ross, Can Pham, Shaw-Yung Shai, Joshua I. Goldhaber, Csilla Fenczik, Chris C. Glembotski, Mark H. Ginsberg, and Joseph C. Loftus Circ Res. 1998;82:1160-1172 Abstract \| Full Text \| PDF
	Increased Protein Kinase C Activity in Myotrophin-Induced Myocyte Growth Parames Sil, Vijaya Kandaswamy, and Subha Sen Circ Res. 1998;82:1173-1188 Abstract \| Full Text \| PDF
	Regulation of Mitochondrial [NADH] by Cytosolic [Ca²⁺] and Work in Trabeculae From Hypertrophic and Normal Rat Hearts Rolf Brandes, Lars S. Maier, and Donald M. Bers Circ Res. 1998;82:1189-1198 Abstract \| Full Text \| PDF
	Ineffective Perfusion-Contraction Matching in Conscious, Chronically Instrumented Pigs With an Extended Period of Coronary Stenosis Raymond K. Kudej, Bijan Ghaleh, Naoki Sato, You-Tang Shen, Sanford P. Bishop, and Stephen F. Vatner Circ Res. 1998;82:1199-1205 Abstract \| Full Text \| PDF
	Simulation Study of Cellular Electric Properties in Heart Failure Leo Priebe and Dirk J. Beuckelmann Circ Res. 1998;82:1206-1223 Abstract \| Full Text \| PDF
	Rapid Communications
	Complement Gene Expression by Rabbit Heart : Upregulation by Ischemia and Reperfusion Koji Yasojima, Kenneth S. Kilgore, Ruth A. Washington, Benedict R. Lucchesi, and Patrick L. McGeer Circ Res. 1998;82:1224-1230 Abstract \| Full Text \| PDF

Spotlight

TOC Spotlight File

Volume 82, Issue 11; June 15, 1998

Editorials

Myocyte Cell Death in the Diseased Heart

Reviews

Apoptosis : Basic Mechanisms and Implications for Cardiovascular Disease

Original Contributions

Role of Apoptosis in the Disappearance of Infiltrated and Proliferated Interstitial Cells After Myocardial Infarction

Effect of Vacuolar Proton ATPase on pHi, Ca2+, and Apoptosis in Neonatal Cardiomyocytes During Metabolic Inhibition/Recovery

Angiotensin II Stimulation In Vitro Induces Hypertrophy of Normal and Postinfarcted Ventricular Myocytes

ß1 Integrins Participate in the Hypertrophic Response of Rat Ventricular Myocytes

Increased Protein Kinase C Activity in Myotrophin-Induced Myocyte Growth

Regulation of Mitochondrial [NADH] by Cytosolic [Ca2+] and Work in Trabeculae From Hypertrophic and Normal Rat Hearts

Ineffective Perfusion-Contraction Matching in Conscious, Chronically Instrumented Pigs With an Extended Period of Coronary Stenosis

Simulation Study of Cellular Electric Properties in Heart Failure

Rapid Communications

Complement Gene Expression by Rabbit Heart : Upregulation by Ischemia and Reperfusion

Spotlight

Effect of Vacuolar Proton ATPase on pH_i, Ca²⁺, and Apoptosis in Neonatal Cardiomyocytes During Metabolic Inhibition/Recovery

ß₁ Integrins Participate in the Hypertrophic Response of Rat Ventricular Myocytes

Regulation of Mitochondrial [NADH] by Cytosolic [Ca²⁺] and Work in Trabeculae From Hypertrophic and Normal Rat Hearts