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miR-1 Overexpression Enhances Ca2+ Release and Promotes Cardiac Arrhythmogenesis by Targeting PP2A Regulatory Subunit B56α and Causing CaMKII-Dependent Hyperphosphorylation of RyR2
Dmitry Terentyev, Andriy E. Belevych, Radmila Terentyeva, Mickey M. Martin, Geraldine E. Malana, Donald E. Kuhn, Maha Abdellatif, David S. Feldman, Terry S. Elton, Sandor Gyorke
https://doi.org/10.1161/CIRCRESAHA.108.181651
Circulation Research. 2009;
Originally published January 8, 2009Dmitry Terentyev
Andriy E. Belevych
Radmila Terentyeva
Mickey M. Martin
Geraldine E. Malana
Donald E. Kuhn
Maha Abdellatif
David S. Feldman
Terry S. Elton

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- miR-1 Overexpression Enhances Ca2+ Release and Promotes Cardiac Arrhythmogenesis by Targeting PP2A Regulatory Subunit B56α and Causing CaMKII-Dependent Hyperphosphorylation of RyR2Dmitry Terentyev, Andriy E. Belevych, Radmila Terentyeva, Mickey M. Martin, Geraldine E. Malana, Donald E. Kuhn, Maha Abdellatif, David S. Feldman, Terry S. Elton and Sandor GyorkeCirculation Research. 2009;, originally published January 8, 2009https://doi.org/10.1161/CIRCRESAHA.108.181651
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- miR-1 Overexpression Enhances Ca2+ Release and Promotes Cardiac Arrhythmogenesis by Targeting PP2A Regulatory Subunit B56α and Causing CaMKII-Dependent Hyperphosphorylation of RyR2Dmitry Terentyev, Andriy E. Belevych, Radmila Terentyeva, Mickey M. Martin, Geraldine E. Malana, Donald E. Kuhn, Maha Abdellatif, David S. Feldman, Terry S. Elton and Sandor GyorkeCirculation Research. 2009;, originally published January 8, 2009https://doi.org/10.1161/CIRCRESAHA.108.181651