Correction for Pepine et al., Circ Res 99 (5) 459-461.

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(Circulation Research. 2006;99:E84.)
© 2006 American Heart Association, Inc.

Correction

An extract of the first 100% of the full text is provided, because this article has no abstract.

In an editorial by Pepine et al. (Circ Res. 2006;99:459-461), ‘Estrogen and Different Aspects of Vascular Disease in Women and Men,‘ the name of the third author was omitted. The correct authorship is Carl J. Pepine, Wilmer W. Nichols and, Daniel F. Pauly. Additionally, the figure was not reproduced correctly, resulting in the erroneous presentation of two symbols. The corrected figure appears below. The corrected article is now available at http://circres.ahajournals.org.

View larger version (27K): [in this window] [in a new window]   Hypothetical construct based on Montague et al suggests that the activational state of estrogen receptor-alpha (ESR1) governs the vascular smooth muscle (SMC) phenotype. Among men with lower ESR1 expression they identified higher levels of differentiation markers compared with women. However activation of ESR1 in SMCs containing low ESR1 expression reduced differentiation markers and promoted apoptosis. This was accomplished by the authors using lentivirus transduction. Conversely, inhibiting ESR1 in SMCs expressing high levels of interfering RNA (SiRNA). The balance between these mechanisms could contribute to vascular remodeling and plaque instability.

The Publisher regrets these errors.

Related Article:

Estrogen and Different Aspects of Vascular Disease in Women and Men

Carl J. Pepine, Wilmer W. Nichols, and Daniel F. Pauly
Circ. Res. 2006 99: 459-461. [Extract] [Full Text] [PDF]