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Circulation Research
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Circulation Research. 2007;101:e1
doi: 10.1161/01.res.0000279319.01839.7d
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(Circulation Research. 2007;101:e1.)
© 2007 American Heart Association, Inc.

Correction

In an article by Lee et al (Circ Res. 2007;100:372–380), the authors regrettably supplied an incorrectly loaded gel for tubulin for Figure 3 panel A. The corrected figure appears here.


Figure 3
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Figure 3. FOXO3a blocked angiotensin II–induced CYR61 expression. A, Serial immunoblot analysis of CYR61, phosphorylated (thus inactivated) native FOXO3a and total native FOXO3a after angiotensin II (10–7 mol/L) stimulation. B, Serial immunoblot analysis of CYR61 protein at 2, 4, and 6 hours after angiotensin II stimulation. C, Corresponding immunohistochemistry of FOXO3a and CYR61 2 hours after angiotensin II stimulation in rat aorta that were pretransfected with Ad-GFP (left), Ad-TM-FOXO3a (middle), and Ad-antisense-FOXO3a (right), respectively. Effective transfection of Ad-TM-FOXO3a and Ad-GFP to rat aorta was confirmed by staining for HA, whereas Ad-antisense-FOXO3a does not contain sequences for HA.





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