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(Circulation Research. 2004;94:1151.)
© 2004 American Heart Association, Inc.

Editorials

In Memoriam

Burton Horowitz

Joseph R. Hume, Mark T. Nelson

From the Department of Pharmacology, University of Nevada School of Medicine, Reno (J.R.H.); and the Department of Pharmacology, University of Vermont, Burlington (M.T.N).

Correspondence to Dr Joseph R. Hume, Department of Pharmacology/318, University of Nevada School of Medicine, Reno, NV 89557. E-mail joeh@med.unr.edu

An extract of the first 250 words of the full text is provided, because this article has no abstract.

Burt Horowitz, close friend, colleague, and valued collaborator of many in the fields of smooth muscle, vascular, and cardiac research, died prematurely at the age of 48 on December 19, 2003. Dr Horowitz was Professor of Physiology & Cell Biology at the University of Nevada School of Medicine (UNSOM) and was a former member of the Circulation Research Editorial Board. He was internationally recognized for his pioneering research on the molecular biology of ion channels in smooth muscle and the cardiovascular system. Burt is survived by his wife, Nancy, and their two beloved daughters, Claire and Stephanie.

Dr Horowitz grew up in Brooklyn, New York, and received a BS degree from Rutgers University in 1976 and a MS degree from California State University, Long Beach, in 1980. He earned a PhD in 1985 in molecular biology with Richard Deonier, followed by postdoctoral training with Robert Farley, both at the University of Southern California. He joined the faculty of UNSOM as an Assistant Professor in Physiology and Cell Biology in 1989.

At the time of Burt’s move to UNSOM, little was known about the molecular basis of smooth muscle excitability. Smooth muscle cells in different organs exhibit unique excitation–contraction properties that underlie their fundamental differences in function. Burt provided groundbreaking research to elucidate the molecular properties of a variety of different types of ion channels in visceral and vascular smooth muscle. He provided seminal information on the cellular and molecular basis for gastrointestinal rhythmicity, including the roles of different types of . . . [Full Text of this Article]