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Circulation Research
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Valvular heart disease

Citations 1-10 of 14 total displayed.

Most recent content (18 Sep 2008):

Articles
Scleraxis Is Required for Cell Lineage Differentiation and Extracellular Matrix Remodeling During Murine Heart Valve Formation In Vivo
Agata K. Levay, Jacqueline D. Peacock, Yinhui Lu, Manuel Koch, Robert B. Hinton, Jr, Karl E. Kadler, and Joy Lincoln
Circ. Res. published September 18, 2008, doi:10.1161/CIRCRESAHA.108.177238 [Abstract]  

Past content (since Jun 2004):

Integrative Physiology
Sympathetic Activation Causes Focal Adhesion Signaling Alteration in Early Compensated Volume Overload Attributable to Isolated Mitral Regurgitation in the Dog
Abdelkarim Sabri, Khadija Rafiq, Rachid Seqqat, Mikhail A. Kolpakov, Ray Dillon, and Louis J. Dell’italia
Circ. Res. 102: 1127 -1136; published online before print as doi:10.1161/CIRCRESAHA.107.163642 [Abstract] [Full text]  

Cellular Biology
Periostin Is Required for Maturation and Extracellular Matrix Stabilization of Noncardiomyocyte Lineages of the Heart
Paige Snider, Robert B. Hinton, Ricardo A. Moreno-Rodriguez, Jian Wang, Rhonda Rogers, Andrew Lindsley, Fang Li, David A. Ingram, Donald Menick, Loren Field, Anthony B. Firulli, Jeffery D. Molkentin, Roger Markwald, and Simon J. Conway
Circ. Res. 102: 752 -760; published online before print as doi:10.1161/CIRCRESAHA.107.159517 [Abstract] [Full text]  

Articles
Sympathetic Activation Causes Focal Adhesion Signaling Alteration in Early Compensated Volume Overload Attributable to Isolated Mitral Regurgitation in the Dog
Abdelkarim Sabri, Khadija Rafiq, Rachid Seqqat, Mikhail A. Kolpakov, Ray Dillon, and Louis J. Dell'italia
Circ. Res. published March 20, 2008, doi:10.1161/CIRCRESAHA.107.163642 [Abstract]  

Articles
Periostin Is Required for Maturation and Extracellular Matrix Stabilization of Noncardiomyocyte Lineages of the Heart
Paige Snider, Robert B. Hinton, Ricardo A. Moreno-Rodriguez, Jian Wang, Rhonda Rogers, Andrew Lindsley, Fang Li, David A. Ingram, Donald Menick, Loren Field, Anthony B. Firulli, Jeffery D. Molkentin, Roger Markwald, and Simon J. Conway
Circ. Res. published February 22, 2008, doi:10.1161/CIRCRESAHA.107.159517 [Abstract]  

Molecular Medicine
Combined Loss of Hey1 and HeyL Causes Congenital Heart Defects Because of Impaired Epithelial to Mesenchymal Transition
Andreas Fischer, Christian Steidl, Toni U. Wagner, Esra Lang, Peter M. Jakob, Peter Friedl, Klaus-Peter Knobeloch, and Manfred Gessler
Circ. Res. 100: 856 -863; published online before print as doi:10.1161/01.RES.0000260913.95642.3b [Abstract] [Full text]  

Articles
Combined Loss of Hey1 and HeyL Causes Congenital Heart Defects Because of Impaired Epithelial to Mesenchymal Transition
Andreas Fischer, Christian Steidl, Toni U. Wagner, Esra Lang, Peter M. Jakob, Peter Friedl, Klaus-Peter Knobeloch, and Manfred Gessler
Circ. Res. published February 15, 2007, doi:10.1161/01.RES.0000260913.95642.3b [Abstract]  

Integrative Physiology
Extracellular Matrix Remodeling and Organization in Developing and Diseased Aortic Valves
Robert B. Hinton, Jr, Joy Lincoln, Gail H. Deutsch, Hanna Osinska, Peter B. Manning, D. Woodrow Benson, and Katherine E. Yutzey
Circ. Res. 98: 1431 -1438; published online before print as doi:10.1161/01.RES.0000224114.65109.4e [Abstract] [Full text]  

Articles
Extracellular Matrix Remodeling and Organization in Developing and Diseased Aortic Valves
Robert B. Hinton, Jr, Joy Lincoln, Gail H. Deutsch, Hanna Osinska, Peter B. Manning, D. Woodrow Benson, and Katherine E. Yutzey
Circ. Res. published April 27, 2006, doi:10.1161/01.RES.0000224114.65109.4e [Abstract]  

Integrative Physiology
Spatial Heterogeneity of Endothelial Phenotypes Correlates With Side-Specific Vulnerability to Calcification in Normal Porcine Aortic Valves
Craig A. Simmons, Gregory R. Grant, Elisabetta Manduchi, and Peter F. Davies
Circ. Res. 96: 792 -799; published online before print as doi:10.1161/01.RES.0000161998.92009.64 [Abstract] [Full text]  

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