Myocardial cardiomyopathy disease

Citations 1-10 of 117 total displayed.

Most recent content (6 Nov 2009):

Integrative Physiology
Role of Ca²⁺/Calmodulin-Stimulated Cyclic Nucleotide Phosphodiesterase 1 in Mediating Cardiomyocyte Hypertrophy: Clint L. Miller, Masayoshi Oikawa, Yujun Cai, Andrew P. Wojtovich, David J. Nagel, Xiangbin Xu, Haodong Xu, Vince Florio, Sergei D. Rybalkin, Joseph A. Beavo, Yiu-Fai Chen, Jian-Dong Li, Burns C. Blaxall, Jun-ichi Abe, and Chen Yan
Circ. Res. 2009;105:956-964; published online before print as doi:10.1161/CIRCRESAHA.109.198515 [Abstract] [Full text]

Integrative Physiology
Dystroglycan Matrix Receptor Function in Cardiac Myocytes Is Important for Limiting Activity-Induced Myocardial Damage: Daniel E. Michele, Zhyldyz Kabaeva, Sarah L. Davis, Robert M. Weiss, and Kevin P. Campbell
Circ. Res. 2009;105:984-993; published online before print as doi:10.1161/CIRCRESAHA.109.199489 [Abstract] [Full text]

Past content (since Mar 2000):

Articles
Conditional Ablation of Nonmuscle Myosin II-B Delineates Heart Defects in Adult Mice: Xuefei Ma, Kazuyo Takeda, Aman Singh, Zu-Xi Yu, Patricia Zerfas, Anthony Blount, Chengyu Liu, Jeffrey A. Towbin, Michael D. Schneider, Robert S. Adelstein, and Qize Wei
Circ. Res. published October 8, 2009, doi:10.1161/CIRCRESAHA.109.200303 [Abstract]

Articles
Dystroglycan Matrix Receptor Function in Cardiac Myocytes Is Important for Limiting Activity-Induced Myocardial Damage: Daniel E. Michele, Zhyldyz Kabaeva, Sarah L. Davis, Robert M. Weiss, and Kevin P. Campbell
Circ. Res. published September 24, 2009, doi:10.1161/CIRCRESAHA.109.199489 [Abstract]

Articles
Role of Ca²⁺/Calmodulin–Stimulated Cyclic Nucleotide Phosphodiesterase 1 in Mediating Cardiomyocyte Hypertrophy: Clint L. Miller, Masayoshi Oikawa, Yujun Cai, Andrew P. Wojtovich, David J. Nagel, Xiangbin Xu, Haodong Xu, Vince Florio, Sergei D. Rybalkin, Joseph A. Beavo, Yiu-Fai Chen, Jian-Dong Li, Burns C. Blaxall, Jun-ichi Abe, and Chen Yan
Circ. Res. published September 24, 2009, doi:10.1161/CIRCRESAHA.109.198515 [Abstract]

Cellular Biology
Heart-Infiltrating Prominin-1⁺/CD133⁺ Progenitor Cells Represent the Cellular Source of Transforming Growth Factor β–Mediated Cardiac Fibrosis in Experimental Autoimmune Myocarditis: Gabriela Kania, Przemyslaw Blyszczuk, Sokrates Stein, Alan Valaperti, Davide Germano, Stephan Dirnhofer, Lukas Hunziker, Christian M. Matter, and Urs Eriksson
Circ. Res. 2009;105:462-470; published online before print as doi:10.1161/CIRCRESAHA.109.196287 [Abstract] [Full text]

Editorials
Expanded Spectrum of Gene Causing Both Hypertrophic Cardiomyopathy and Dilated Cardiomyopathy: Sachio Morimoto
Circ. Res. 2009;105:313-315, doi:10.1161/CIRCRESAHA.109.204180 [Full text]

Clinical Research
Identification and Functional Characterization of Cardiac Troponin I As a Novel Disease Gene in Autosomal Dominant Dilated Cardiomyopathy: Sebastian Carballo, Paul Robinson, Robyn Otway, Diane Fatkin, Jan D.H. Jongbloed, Nicolaas de Jonge, Edward Blair, J. Peter van Tintelen, Charles Redwood, and Hugh Watkins
Circ. Res. 2009;105:375-382; published online before print as doi:10.1161/CIRCRESAHA.109.196055 [Abstract] [Full text]

Reports
Evidence From Human Myectomy Samples That MYBPC3 Mutations Cause Hypertrophic Cardiomyopathy Through Haploinsufficiency: Steven Marston, O‘Neal Copeland, Adam Jacques, Karen Livesey, Victor Tsang, William J. McKenna, Shapour Jalilzadeh, Sebastian Carballo, Charles Redwood, and Hugh Watkins
Circ. Res. 2009;105:219-222; published online before print as doi:10.1161/CIRCRESAHA.109.202440 [Abstract] [Full text]

Integrative Physiology
Cardiomyocyte-Specific Loss of Neurofibromin Promotes Cardiac Hypertrophy and Dysfunction: Junwang Xu, Fraz A. Ismat, Tao Wang, Min Min Lu, Nicole Antonucci, and Jonathan A. Epstein
Circ. Res. 2009;105:304-311; published online before print as doi:10.1161/CIRCRESAHA.109.201509 [Abstract] [Full text]

Subspecialty Collections Home

Show Myocardial cardiomyopathy disease
collections from other journals.