|
Heart failure - basic studies
Citations 271-280 of 418 total displayed.
|
Past content
(since Jan 1999):
|
- Articles
Brain Natriuretic Peptide Is Produced in Cardiac Fibroblasts and Induces Matrix Metalloproteinases
- Toshihiro Tsuruda, Guido Boerrigter, Brenda K. Huntley, Josh A. Noser, Alessandro Cataliotti, Lisa C. Costello-Boerrigter, Horng H. Chen, and John C. Burnett, Jr
Circ. Res. published November 7, 2002, doi:10.1161/01.RES.0000046234.73401.70
[Abstract]
- Articles
Ventricular Hypertrophy Plus Neurohumoral Activation Is Necessary to Alter the Cardiac -Adrenoceptor System in Experimental Heart Failure
- Kirsten Leineweber, Katja Brandt, Beate Wludyka, Anja Beilfuß, Klaus Pönicke, Ingrid Heinroth-Hoffmann, and Otto-Erich Brodde
Circ. Res. published November 7, 2002, doi:10.1161/01.RES.0000045088.59360.B7
[Abstract]
- Articles
Abnormal Ca2+ Release, but Normal Ryanodine Receptors, in Canine and Human Heart Failure
- Ming Tao Jiang, Andrew J. Lokuta, Emily F. Farrell, Matthew R. Wolff, Robert A. Haworth, and Héctor H. Valdivia
Circ. Res. published October 24, 2002, doi:10.1161/01.RES.0000043663.08689.05
[Abstract]
- Editorials
L-Type Ca2+ Channels Gaining Respect in Heart Failure
- Timothy J. Kamp and Jia-Qiang He
Circ. Res. 2002;91:451-453, doi:10.1161/01.RES.0000035346.21625.4A
[Full text]
- Cellular Biology
L-Type Ca2+ Channel Density and Regulation Are Altered in Failing Human Ventricular Myocytes and Recover After Support With Mechanical Assist Devices
- Xiongwen Chen, Valentino Piacentino, III, Satoshi Furukawa, Bruce Goldman, Kenneth B. Margulies, and Steven R. Houser
Circ. Res. 2002;91:517-524; published online before print as doi:10.1161/01.RES.0000033988.13062.7C
[Abstract]
[Full text]
- Cellular Biology
Increased Exchange Current but Normal Ca2+ Transport via Na+-Ca2+ Exchange During Cardiac Hypertrophy After Myocardial Infarction
- Ana Maria Gómez, Beat Schwaller, Hartmut Porzig, Guy Vassort, Ernst Niggli, and Marcel Egger
Circ. Res. 2002;91:323-330; published online before print as doi:10.1161/01.RES.0000031384.55006.DB
[Abstract]
[Full text]
- Editorials
Hypoxia, BNip3 Proteins, and the Mitochondrial Death Pathway in Cardiomyocytes
- Michael T. Crow
Circ. Res. 2002;91:183-185, doi:10.1161/01.RES.0000030195.38795.CF
[Full text]
- Cellular Biology
Inducible Expression of BNIP3 Provokes Mitochondrial Defects and Hypoxia-Mediated Cell Death of Ventricular Myocytes
- Kelly M. Regula, Karen Ens, and Lorrie A. Kirshenbaum
Circ. Res. 2002;91:226-231; published online before print as doi:10.1161/01.RES.0000029232.42227.16
[Abstract]
[Full text]
- Articles
Increased Exchange Current but Normal Ca2+ Transport via Na+-Ca2+ Exchange During Cardiac Hypertrophy After Myocardial Infarction
- Ana Maria Gómez, Beat Schwaller, Hartmut Porzig, Guy Vassort, Ernst Niggli, and Marcel Egger
Circ. Res. published August 1, 2002, doi:10.1161/01.RES.0000031384.55006.DB
[Abstract]
- Cellular Biology
Actin Capping Protein: An Essential Element in Protein Kinase Signaling to the Myofilaments
- W. Glen Pyle, Marilyn C. Hart, John A. Cooper, Marius P. Sumandea, Pieter P. de Tombe, and R. John Solaro
Circ. Res. 2002;90:1299-1306; published online before print as doi:10.1161/01.RES.0000024389.03152.22
[Abstract]
[Full text]
|
|