Arrythmias-basic studies

Citations 141-150 of 456 total displayed.

Past content (since Feb 1999):

Cellular Biology
Reducing Ryanodine Receptor Open Probability as a Means to Abolish Spontaneous Ca²⁺ Release and Increase Ca²⁺ Transient Amplitude in Adult Ventricular Myocytes: L.A. Venetucci, A.W. Trafford, M.E. Díaz, S.C. O’Neill, and D.A. Eisner
Circ. Res. 2006;98:1299-1305; published online before print as doi:10.1161/01.RES.0000222000.35500.65 [Abstract] [Full text]

Articles
14-3-3 Is a Regulator of the Cardiac Voltage-Gated Sodium Channel Nav1.5: Marie Allouis, Françoise Le Bouffant, Ronald Wilders, David Peroz, Jean-Jacques Schott, Jacques Noireaud, Herve Le Marec, Jean Merot, Denis Escande, and Isabelle Baró
Circ. Res. published May 25, 2006, doi:10.1161/01.RES.0000229244.97497.2c [Abstract]

Articles
Selectivity of Connexin 43 Channels Is Regulated Through Protein Kinase C-Dependent Phosphorylation: Jose F. Ek-Vitorin, Timothy J. King, Nathanael S. Heyman, Paul D. Lampe, and Janis M. Burt
Circ. Res. published May 18, 2006, doi:10.1161/01.RES.0000227572.45891.2c [Abstract]

Cellular Biology
Abnormal Interactions of Calsequestrin With the Ryanodine Receptor Calcium Release Channel Complex Linked to Exercise-Induced Sudden Cardiac Death: Dmitry Terentyev, Alessandra Nori, Massimo Santoro, Serge Viatchenko-Karpinski, Zuzana Kubalova, Inna Gyorke, Radmila Terentyeva, Srikanth Vedamoorthyrao, Nico A. Blom, Giorgia Valle, Carlo Napolitano, Simon C. Williams, Pompeo Volpe, Silvia G. Priori, and Sandor Gyorke
Circ. Res. 2006;98:1151-1158; published online before print as doi:10.1161/01.RES.0000220647.93982.08 [Abstract] [Full text]

Cellular Biology
Angiotensin II and Stretch Activate NADPH Oxidase to Destabilize Cardiac Kv4.3 Channel mRNA: Chaoming Zhou, Chandra Ziegler, Lori A. Birder, Alexandre F.R. Stewart, and Edwin S. Levitan
Circ. Res. 2006;98:1040-1047; published online before print as doi:10.1161/01.RES.0000218989.52072.e7 [Abstract] [Full text]

Cellular Biology
KCNQ1 Assembly and Function Is Blocked by Long-QT Syndrome Mutations That Disrupt Interaction With Calmodulin: Smita Ghosh, Deborah A. Nunziato, and Geoffrey S. Pitt
Circ. Res. 2006;98:1048-1054; published online before print as doi:10.1161/01.RES.0000218863.44140.f2 [Abstract] [Full text]

Cellular Biology
Calmodulin Is Essential for Cardiac I_KS Channel Gating and Assembly: Impaired Function in Long-QT Mutations: Liora Shamgar, Lijuan Ma, Nicole Schmitt, Yoni Haitin, Asher Peretz, Reuven Wiener, Joel Hirsch, Olaf Pongs, and Bernard Attali
Circ. Res. 2006;98:1055-1063; published online before print as doi:10.1161/01.RES.0000218979.40770.69 [Abstract] [Full text]

Articles
Reducing Ryanodine Receptor Open Probability As a Means to Abolish Spontaneous Ca²⁺ Release and Increase Ca²⁺ Transient Amplitude in Adult Ventricular Myocytes: L. A. Venetucci, A. W. Trafford, M. E. Díaz, S. C. O’Neill, and D. A. Eisner
Circ. Res. published April 13, 2006, doi:10.1161/01.RES.0000222000.35500.65 [Abstract]

Articles
Abnormal Interactions of Calsequestrin With the Ryanodine Receptor Calcium Release Channel Complex Linked to Exercise-Induced Sudden Cardiac Death: Dmitry Terentyev, Alessandra Nori, Massimo Santoro, Serge Viatchenko-Karpinski, Zuzana Kubalova, Inna Gyorke, Radmila Terentyeva, Srikanth Vedamoorthyrao, Nico A. Blom, Giorgia Valle, Carlo Napolitano, Simon C. Williams, Pompeo Volpe, Silvia G. Priori, and Sandor Gyorke
Circ. Res. published April 6, 2006, doi:10.1161/01.RES.0000220647.93982.08 [Abstract]

Cellular Biology
Electrotonic Modulation of Cardiac Impulse Conduction by Myofibroblasts: Michele Miragoli, Giedrius Gaudesius, and Stephan Rohr
Circ. Res. 2006;98:801-810; published online before print as doi:10.1161/01.RES.0000214537.44195.a3 [Abstract] [Full text]

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